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COVID 19: “Autoantibodies related to systemic autoimmune rheumatic diseases in severely ill patients with COVID-19”

Hence my testing definitively positive for Hashimoto’s Thyroiditis via the Thyroid Peroxidase test. I also had rheumatic arthritis symptoms in fingers and knees, since resolved... well, mostly.

I’ll re-test in a few months to see if the antibody levels have dropped.

Autoantibodies related to systemic autoimmune rheumatic diseases in severely ill patients with COVID-19

Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is the cause of COVID-19, which has affected more than 6 million people worldwide causing more than 400 000 deaths. The disease affects predominantly the upper and lower respiratory tracts causing severe pulmonary disease which often evolves to a multiorgan systemic disease...


The small sample size did not allow procurement of any statistically significant clinicolaboratory associations. Despite this and the lack of preinfection serological data, the presence of several systemic autoimmune reactivities in almost 70% of the patients suggests a post-SARS-CoV-2 or para-SARS-CoV-2 infectious autoimmune activation. This is not surprising, as cytokines present in the cytokine storm, for example, interleukin-6, can drive autoinflammatory reactions and autoimmunity, probably via pre-existing natural B cell clones or molecular mimicry. The possible autoimmune mechanism merits further investigation, therefore an autoimmune surveillance in larger cohorts is necessary to investigate possible mechanisms of COVID-19 perpetuation and to inform ongoing convalescence plasma therapeutic trials.7 8 As several of the reported autoantibodies are disease markers and can be pathogenic, one should be cautious before transferring autoantibodies, along with neutralising antibodies, into severely affected patients.

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